While there is a clear genetic predisposition for some individuals to develop autoimmune disorders, there are also significant environmental interactions, with a near epidemic rise in autoimmune disorders and food allergies in the industrialized world occurring in less than fifty years. Access to clean water, safe food, sanitation, and modern medical care in the last century has led to a near elimination of parasitic infections in modern cultures. Unfortunately, improved sanitation has also been closely associated with an increase in hypersensitivity and autoimmune disorders such as Multiple Sclerosis, Crohn’s, and peanut allergies. It should be noted that tropical and subtropical parts of the world with poor sanitation have high numbers of people with ongoing parasitic infection and extremely low incidences of inflammatory diseases. Additionally when immigrants relocate to the western world with its pristine sanitation, the next generation develops autoimmune disorders and food allergies similar to the industrialized world.
The “hygiene hypothesis,” suggests that parasitic worms fine tune the human immune systems and protect against allergies and autoimmune diseases. Parasites have co-evolved to manipulate their environments so they can live and reproduce without causing the illness or death of their host. By mediating the immune system’s attack against them, parasites can live and reproduce comfortably. Parasites release substances that interact with the host’s chemistry, and the human immune system has slowly adapted to the presence of certain intestinal parasites. By removing the worms and the inhibitory effect that they have on inflammation, the human body can overproduce inflammatory agents, resulting in autoimmune disease.
Autoimmune reactions occur when the immune system launches an attack at something that is not harmful, either a substance in the environment or the individual’s own tissues. Exposure to certain organisms in our natural environment teaches the human immune system which things it needs to respond to and which things it can safely ignore. Exposure to helminthic parasites triggers the fine-tuning of the immune system and teaches it to tolerate the parasite, as well as some overlapping antigens seen in Multiple Sclerosis, Crohn’s and peanut allergies. If the immune system is never exposed to helminths and other fecal microorganisms, it doesn’t mature properly and resulting in an exaggerated response, and development of allergies and autoimmune diseases.
Other autoimmune mediated diseases that may be reduced by parasitic infections include Alzheimer’s disease, asthma, atherosclerosis, some cancers, eczema, hay fever, Parkinson’s disease, type one diabetes, celiac disease, rheumatoid arthritis, and ulcerative colitis.
The use of helminthic therapy with hookworm and TSO is being currently investigated. Data suggests that Multiple Sclerosis, Crohn’s and severe peanut allergies all benefit from induced parasitic infections.
Obesity is among the most important medical problems in the United States today. Currently, 1 in 4 children and 1 in 2 adults are overweight, and prevalence rates that have increased by 50% since the 1960s. The Federal government and various official medical agencies, at the behest of grain producers, have advocated decreasing intake of total fat, while increasing consumption of “complex carbohydrate.” Consumption of carbohydrates has increased over the years, and the nation’s levels of obesity, Type 2 diabetes and heart disease have dramatically risen. Americans, on average, eat 250 to 300 grams of carbs a day, accounting for about 55% of their caloric intake.
All carbohydrates (a category including sugars) convert to sugar in the blood, and the more refined the carbs are, the quicker the conversion. When you eat a glazed doughnut or a serving of mashed potatoes, it turns into blood sugar very quickly. To manage the blood sugar, the pancreas produces insulin, which pushes glucose from the blood stream into cell to be used for energy conversion or storage.
When cells become more resistant to those insulin instructions, the pancreas needs to make more insulin to push the same amount of glucose into cells. As people become insulin resistant, carbs become a bigger challenge for the body. When the pancreas gets exhausted and can’t produce enough insulin to keep up with the glucose in the blood, diabetes develops
The first sign of insulin resistance is a condition called metabolic syndrome — a red flag for impending diabetes and heart disease. Metabolic syndrome (found in nearly 1/4 of adults) is diagnosed when people have three or more of the following:
high blood triglycerides (more than 150 mg)
high blood pressure (over 135/85)
central obesity (a waist circumference in men of more than 40 inches and in women, more than 35 inches)
low HDL cholesterol (under 40 in men, under 50 in women)
elevated fasting glucose.
Glycemic Index measures the “effect of food on blood glucose levels.” It is a ranking of foods based on the how quickly the blood sugar levels will increase after ingestion. A low glycemic food gives a slow increase in blood sugar levels. A high glycemic index food gives a more rapid rise in blood sugar levels.
GI is specifically defined as the measurable glucose response curve after consumption of 50 g carbohydrate from a test food, divided by the response after consumption of 50 g glucose.
The GI for glucose would be defined as 100.
High GIs are above 50
Intermediate GIs range between 35 and 50
Low GIs are below or equal to 35
[learn_more caption=”High Glycemic Index Foods (GI>50)”]
In general, refined grain products and potato have a high GI, exceeding that of table sugar by up to 50%, whereas most vegetables, fruits and legumes have a low GI. Other factors including carbohydrate type, fiber, protein, fat, food form and method of preparation, determine the GI of a particular food.
According to data from the Department of Agriculture, >80% of the carbohydrate currently consumed by children ages 2–18 has a GI equal to or greater than that of table sugar. Moreover, carbohydrate absorption rate (and therefore GI) is increased after a low fat meal because fat acts to delay gastric emptying.
The rapid absorption of glucose from the high GI meal results in a high insulin secretion; which promotes uptake of glucose in muscle, liver and fat tissue and inhibits fat breakdown (lipolysis). In the post absorptive period, a transient hypoglycemia ensues, with blood sugars falling below normal due to high insulin, resulting in hunger and agitation. In some individuals, this may cause tremendous anxiety, which may create a feedback loop of carbohydrate addiction. Experimental evidence also suggests that elevated insulin levels, even just for 48–72-h period (in the presence of normal or reduced blood sugar levels) decreases insulin sensitivity in healthy subjects creating a diabetic effect of insulin resistance.
Without a doubt, high GI foods elicit (calorie for calorie) higher insulin levels than low GI foods. In humans, high acute insulin secretion after intravenous glucose tolerance tests predicts weight gain. High insulin levels also reduce Growth Hormone levels, which may reduce metabolic rate. Hormonal responses to a high GI diet stimulate hunger and favor storage of fat, which promotes excessive weight gain.
The LiveHealthProtocol dietary recommendation is designed to lower the insulin response to ingested carbohydrate (low GI), which improves access to stored metabolic fuels, decreases hunger, and promotes weight loss. The LiveHealthProtocol recommends abundant quantities of vegetables, and fruits, moderate amounts of protein and healthful fats, and decreased intake of refined grain products, potato and concentrated sugars.
Milk products (whey protein) have a GI which is low, but have paradoxic high insulinemic index (release high amounts of insulin). Milk products appear insulinotropic as judged from 3-fold to 6-fold higher insulinemic indexes than expected from the corresponding glycemic indexes. So even if you are consuming a low GI milk product, from the insulin standpoint it is a very high load.
Starchy fruits increase their Glycemic Index depending on ripeness. Green bananas have low GI of 40 but when they are ripen it will raise to 65.
Glycemic Load [GL] relates the GI to the amount of carbohydrate eaten in a normal serving or in 100 grams. It measures the total amount of carbohydrate, and is decreased by fiber consumption.
As your Body Mass Index [BMI] increases, so does your risk of death. But is BMI the best tool to determine your health status?
Calculated from your weight and height, BMI provides a general gauge of body fatness. BMI is a correlation between an individual’s height and weight, but it does not distinguish between fat weight and muscle weight.
Figure your Body Mass Index using either your bioempedence scale, your body fat calipers or the following simple formula:
BMI=Weight in pounds x 703, divided by height in inches x height in inches
BMI=[Weight x 703]/height2
Weight in pounds = 150
Height in inches = 64
150 x 703 = 105,450
64 x 64 = 4096
105,450/4096 = 25.74
BMI = 25.74%
Your BMI number can fall into one of four adult categories:
Below 18.5 = Underweight
18.5 – 24.9+ = Normal
25.0 – 29.9 = Overweight
30.0 and above = Obese
As your BMI increases, so does your risk of death from many possible causes, such as ischemic heart disease, stroke and various types of cancers. Based upon a study published in the online March 2009 Lancet, a collaborative analysis of 57 studies found that BMI is a “reasonably good measure” of general adiposity – body fat.
Researchers reported the lowest mortality was in those with a BMI of 22.5 to 25 kg/m2. As BMI slightly increases, the mortality rate begins to skyrocket. each 5 kg/m2 higher BMI was associated with about 30% higher all-cause mortality (40% for vascular; 60–120% for diabetic, renal, and hepatic; 10% for neoplastic; and 20% for respiratory and for all other mortality). In other words, as BMI increased overall death rate increased.
Having an above normal range BMI can result in these health conditions:
Coronary heart disease
People frequently state that muscle weighs more than fat. Its simply not true, a pound of muscle weighs exactly the same as a pound of fat. It just so happens that fat occupies nearly 18% more volume for the same pound weight, and muscle tends to adhere to tightly to skeletal structures, whereas fat drapes loosly. Because BMI fails to distinguish between fat and muscle, because it is weight based, a better way of looking at this was needed, hence a calculation of actual percentage fat by bioimpedance scale or skin calipers or more scientifically with DEXA scan.
Danger also lurks behind that expanding waistline: highly active, toxic abdominal fat heightens your disease risk. Check your waist girth with a tape measure, starting at the top of the hipbone. Measure at your greatest waist area, keeping the tape measure even, not pulling too tightly. A total of over 40 inches for men or over 35 inches for women means it’s high time to get serious about taking control of your health and future. The abdominal girth or waist measurement is a quick tool in determining your cardiac risk, it looks more at the volume of fat in the abdomen rather than the weight.
The waist to hip ratio is an important tool that helps you determine your overall health risk. People with more weight around their waist are at greater risk of lifestyle related diseases such as heart disease and diabetes than those with weight around their hips. It is a simple and useful measure of fat distribution.
Use a measuring tape to check the waist and hip measurements. - Measure your hip circumference at it’s widest part. - Measure your Waist Circumference at the belly button or just above it.
Simply take your waist measurement / your hip measurement; for example:
Waist to hip ratio:
Waist circumference / hip circumference
Ratio: 32/30 = 1.07
MALE FEMALE: Health risk based on ratio only
.95 or below .80 or below Low Risk
.96 – 1.0 .81-.85 Moderate Risk
1.0 + .85 + High Risk
Perhaps exercise tolerance and maximal exercise capacity by measuring METs may be the best indicator of body health and longevity, a functional test of exercise capacity rather than a weight test or a size test.
If you are truly interested in losing abdominal fat, check out our mdHCG protocol for a guaranteed solution.
There are three types of fat commonly seen observed in the abdomen
Subcutaneous Fat, which is fat immediately below the skin but outside the abdominal cavity.
Retroperitoneal or Structural Fat separating organ and providing cushioning during movement.
Visceral abdominal fat (found inside the abdominal cavity). Most of that fat is found on the greater omentum—a large apron-like sheet that drapes over all the organs. Some people are prone to storing their body fat in their abdomen as part of this greater omentum. As a result, large, globule clumps of fat attach to the omentum, where they release toxic materials into the venous drainage and onto the bloodstream, ultimately causing adverse effects. This kind of fat releases adipokines, harmful chemicals that can cause Alzheimer’s, type 2 diabetes, insulin resistance, high blood pressure, and increased risk of cancer.
Reports have demonstrated that being overweight or obese (particular with a centralized distribution of adiposity) puts you in a higher-risk category for chronic conditions, from insulin resistance, type 2 diabetes, high blood pressure, high cholesterol, stroke, heart attack, congestive heart failure, gallstones, gout, osteoarthritis, sleep apnea and a liver disease called nonalcoholic fatty liver disease (NAFLD).
It is interesting to note that the middle-age bulge increase your chances of having
Alzheimer’s or other types of dementia in your senior years by nearly three times, even though it’s not your brain that stores the fat. This may be related to:
Toxic metabolites produced by the visceral abdominal fat, such as xenoestrogens which suppress testosterone formation, leading to poor blood vessel elasticity and high blood pressure
Storage of fat soluble toxins, such as food borne pesticides and herbicides, acquired from the environment which then slowly leach out.
Toxic belly fat is thought to promote amyloid accumulation by increasing chronic inflammation. Amyloid is an abnormal protein, which deposits in the body’s tissues or in more organ-specific areas, such as in the pancreas (type 2 diabetes) or central nervous system (Alzheimer’s, Parkinson’s, Huntington disease).
Visceral abdominal fat (belly fat) is the last reservoir people get rid of when they’re losing body fat—and it’s the first place they store it when they gain back body fat.
To date, there are no ways to specifically target that fat, but there are things you can do to work your way down to healthier body fat levels (16% for men, under 22% for women).
A low-glycemic nutrition eating plan with specific nutritional supplementation can help you optimize your health potential and reduce belly fat. Consistent high intensity interval exercise can increase metabolic rates, lower heart disease risk and improve body composition, muscle endurance, flexibility, cardiovascular endurance, core strength/stability and posture. These effects are not purely related to the energy burned due to exercise, but also due to the significant endocrine [hormonal changes] induced by resistance exercise leading to increased Growth Hormone, Thyroid Hormone, and Testosterone production.
More specifically, resistance training helps you lose abdominal fat and overall body fat while lowering cholesterol: Weight training is the best way to burn fat; it’s more effective for losing weight than aerobic activity because it burns calories while you’re exercising and later at rest due to the hormonal changes and muscular regeneration.
If you are truly interested in losing abdominal fat, check out our mdHCG protocol for a guaranteed solution.